Regulation of spinal motoneuron differentiation by the combined action of sonic hedgehog and and neurotrophin 3
Details
Publication Year 1999-09,Volume 26,Issue #9,Page 746-748
Journal Title
CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY
Publication Type
Journal Article
Abstract
1, The development of ventral cell types in the spinal cord, including motor neurons, requires the growth factor Sonic hedgehog (Shh), However, it is still unknown whether Shh acts directly on precursors to induce these cell types and whether additional factors are required for induction. 2, To further investigate the precise role of Shh in spinal motoneuron development, we used low-density cultures of murine spinal cord precursor cells, 3, Although purified Shh stimulated neuronal differentiation, it did not increase the proportion of neurons expressing the motoneuron marker Islet-1 (Isl-1), In contrast, purified Shh induced Isl-l expression in neural tube explants, suggesting that additional neural tube-derived factors are required to induce motoneuron differentiation. 4, A factor implicated in motoneuron development, neurotrophin 3 (NT3), had no effect on Isl-1 expression; however in combination with Shh, it induced Isl-1 expression in the majority of neurons in the low-density cultures. Furthermore, in explant cultures, Shh-induced Isl-1 expression was blocked by an anti-NT3 antibody, 5, In addition, previous studies have shown expression of NT3 in the region of motoneuron differentiation and the loss of spinal fusimotor neurons in NT3-knockout animals. 6, Taken together, these findings suggest that Shh can act directly on spinal cord precursors to promote neuronal differentiation, but induction of Isl-1 expression and motor differentiation requires additional factors, including NT3.
Publisher
BLACKWELL SCIENCE ASIA
Keywords
MOTOR-NEURON INDUCTION; FLOOR PLATE; POLARIZING ACTIVITY; EXPRESSION; NOTOCHORD; ORGANIZATION; REQUIREMENT; RECEPTOR; CELLS; MOUSE
Terms of Use/Rights Notice
Refer to copyright notice on published article.


Creation Date: 1999-09-01 12:00:00
An error has occurred. This application may no longer respond until reloaded. Reload 🗙