Expression of receptor tyrosine kinase Axl and its ligand Gas6 in rheumatoid arthritis - Evidence for a novel endothelial cell survival pathway
Details
Publication Year 1999-04,Volume 154,Issue #4,Page 1171-1180
Journal Title
AMERICAN JOURNAL OF PATHOLOGY
Publication Type
Journal Article
Abstract
Angiogenesis and synovial cell hyperplasia are characteristic features of rheumatoid arthritis CRA), Many growth and survival factors use receptors belonging to the tyrosine kinase family that share conserved motifs within the intracellular catalytic domains. To understand further the molecular basis of cellular hyperplasia in RE, we have used degenerate primers based on these moths and RNA obtained from the synovium of a patient with RA. to perform reverse transcriptase-polymerase chain. reaction. We report detection of the receptor tyrosine kinase (RTK) Axl in Ri synovium and we document the expression pattern of Axl in capillary endothelium, in vascular smooth muscle cells of arterioles and veins, and in a subset of synovial cells in RA synovial tissue. Gas6 (for growth arrest-specific gene 6), which is a ligand for Axl and is related to the coagulation factor protein S, was found in synovial fluid and tissue from patients with RA and osteoarthritis. Axl expression and function was studied in human umbilical vein endothelial cells (HUVECs). Gas6 bound to HUVECs; soluble Axl inhibited this binding. Exogenous Gas6 protected HUVECs from apoptosis in response to growth factor withdrawal and from TNF alpha-mediated cytotoxicity, These findings may reveal a new aspect of vascular physiology, which may also be relevant to formation and maintenance of the abnormal vasculature in the rheumatoid synovium.
Publisher
AMER SOC INVESTIGATIVE PATHOLOGY, INC
Keywords
ARREST-SPECIFIC GENE-6; VASCULAR SMOOTH-MUSCLE; GROWTH-ARREST; PROTEIN-S; SPLICE VARIANT; UFO RECEPTOR; IDENTIFICATION; ADHESION; PRODUCT; ANGIOGENESIS
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Creation Date: 1999-04-01 12:00:00
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