Apoptosis in the development and treatment of cancer
Author(s)
Gerl, R; Vaux, DL;
Details
Publication Year 2005-02,Volume 26,Issue #2,Page 263-270
Journal Title
CARCINOGENESIS
Publication Type
Journal Article
Abstract
Our somatic cells are born by mitosis and almost all will die by apoptosis, a physiological process of cellular suicide. Cancers can occur when this balance is disturbed, either by an increase in cell proliferation or a decrease in cell death. The goal of cancer therapy is to promote the death of cancer cells without causing too much damage to normal cells. Our knowledge of the mechanisms of apoptosis has enhanced our understanding of how some cancers originate and progress. It has also revealed that existing cancer therapies can work in two ways, by induction of apoptosis as well as by direct toxicity. In some cases resistance to apoptosis may explain why cancer therapies fail. Novel treatments designed to exploit our knowledge of apoptotic mechanisms are under development to promote apoptosis of cancer cells and limit concurrent death of normal cells.
Publisher
OXFORD UNIV PRESS
Keywords
PROGRAMMED CELL-DEATH; SMALL-MOLECULE ANTAGONISTS; MYC-INDUCED APOPTOSIS; BCL-2 FAMILY; C-MYC; TRANSGENIC MICE; MALT LYMPHOMA; CHEMOTHERAPEUTIC-AGENTS; PROAPOPTOTIC ACTIVITY; BH3-ONLY PROTEINS
Terms of Use/Rights Notice
Refer to copyright notice on published article.


Creation Date: 2005-02-01 12:00:00
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