Tandem LIM domains provide synergistic binding in the LMO4 : Ldb1 complex
Details
Publication Year 2004-09-15,Volume 23,Issue #18,Page 3589-3598
Journal Title
EMBO JOURNAL
Publication Type
Journal Article
Abstract
Nuclear LIM-only (LMO) and LIM-homeodomain (LIM-HD) proteins have important roles in cell fate determination, organ development and oncogenesis. These proteins contain tandemly arrayed LIM domains that bind the LIM interaction domain ( LID) of the nuclear adaptor protein LIM domain-binding protein-1 (Ldb1). We have determined a high-resolution X-ray crystal structure of LMO4, a putative breast oncoprotein, in complex with Ldb1-LID, providing the first example of a tandem LIM: Ldb1-LID complex and the first structure of a type-B LIM domain. The complex possesses a highly modular structure with Ldb1-LID binding in an extended manner across both LIM domains of LMO4. The interface contains extensive hydrophobic and electrostatic interactions and multiple backbone-backbone hydrogen bonds. A mutagenic screen of Ldb1-LID, assessed by yeast two-hybrid and competition ELISA analysis, identified key features at the interface and revealed that the interaction is tolerant to mutation. These combined properties provide a mechanism for the binding of Ldb1 to numerous LMO and LIM-HD proteins. Furthermore, the modular extended interface may form a general mode of binding to tandem LIM domains.
Publisher
NATURE PUBLISHING GROUP
Keywords
CELL ACUTE-LEUKEMIA; PROTEIN LMO2; ONLY PROTEIN; MACROMOLECULAR STRUCTURES; TRANSCRIPTION FACTORS; HOMEODOMAIN ACTIVITY; INTERACTOR NLI; HOMEOBOX GENE; LDB1; MICE
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Creation Date: 2004-09-15 12:00:00
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