Impaired "outside-in" integrin alpha(IIb)beta(3) signaling and thrombus stability in TSSC6-deficient mice
Details
Publication Year 2006-09-15,Volume 108,Issue #6,Page 1911-1918
Journal Title
BLOOD
Publication Type
Journal Article
Abstract
We investigated the role of the hematopoietic-specific tetraspanin superfamily member, TSSC6, in platelet function using wild-type mice and TSSC6-deficient mice. TSSC6 is expressed on the surface of murine platelets and is up-regulated by thrombin stimulation, indicating an intracellular pool of TSSC6. Immunoprecipitation/Western blot studies reveal a constitutive physical association of TSSC6 with the integrin alpha(IIb)beta(3) complex under strong detergent conditions. In vivo evaluation of hemostasis by tail bleeding revealed increased bleeding time, volume of blood lost, and evidence of tail rebleeds in TSSC6 null mice, indicating unstable hemostasis. Using ex vivo techniques, we showed that TSSC6-deficient platelets exhibited impaired kinetics of clot retraction, platelet aggregation at lower doses of PAR-4, and collagen and platelet spreading on fibrinogen in the presence of normal integrin alpha(IIb)beta(3) expression. TSSC6-deficient platelets showed normal alpha granule secretion, normal "inside-out" integrin alpha(IIb)beta(3) signaling (fluorescein isothiocyanate [FITC]-fibrinogen and JON/A binding), and normal platelet adhesion on fibrinogen. Furthermore, we show that absence of platelet TSSC6 affects the secondary stability of arterial thrombi in vivo upon vascular injury. These data demonstrate that TSSC6 appears to regulate integrin alpha(IIb)beta(3) "outside-in" signaling events in platelets and is necessary for stability of arterial thrombi in vivo.
Publisher
AMER SOC HEMATOLOGY
Keywords
ANTI-CD9 MONOCLONAL-ANTIBODY; SUPERFAMILY MEMBER CD151; TETRASPANIN SUPERFAMILY; PLASMA-MEMBRANE; PROLIFERATIVE RESPONSES; CD9-DEFICIENT MICE; PLATELET-FUNCTION; MOUSE; CD81; CD9
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Creation Date: 2006-09-15 12:00:00
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