Reacting to reductive stress at the mitochondrial import gate
Author(s)
Callegari, S;
Journal Title
Trends in Cell Biology
Publication Type
Jan 13 epub
Abstract
A byproduct of mitochondrial energy production is the generation of reactive oxygen species (ROS). Too much ROS is toxic, but ROS deficiency is equally deleterious (reductive stress). In a recent study, McMinimy et al. uncovered a ubiquitin proteasome-mediated mechanism at the translocase of the outer membrane (TOM) complex, which senses ROS depletion and adjusts mitochondrial protein import accordingly.
Publisher
Cell Press
Keywords
TOM complex; mitochondrial import; proteasome; reactive oxygen species; reductive stress; ubiquitin
Research Division(s)
Ubiquitin Signalling
PubMed ID
39809643/
Terms of Use/Rights Notice
Refer to copyright notice on published article.


Creation Date: 2025-02-07 11:22:04
Last Modified: 2025-02-07 11:24:04
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